Persistent Organic Pollutants Could Cause Type 2 Diabetes
An extraordinary attempt has been stressed upon to investigate an association between the ‘Environmental Pollution (EP) ‘and ‘Type-2 Diabetes’. Scientists from the “Cambridge University” are making serious attempts shed more light on the least unexplored relationship between the two of them.
Writing in a research article in the most recent edition of the journal “Lancet”, Drs. Oliver Jones and Julian Griffin highlighted the need to research the possible link between the ‘Persistent Organic Pollutants (POPs), a group which includes many pesticides, and insulin resistance. A most probable relationship between the two could possibly lead to adult onset diabetes.
The researchers made special mentioning of their peer based reviewed research and on the basis of the results obtained they demonstrated that there exists a very linear bond type relationship between the levels of POPs in blood, particularly the ‘organo-chlorine compounds’, and the risk of ‘Type-2 Diabetes’.
Scientists do not have any satisfying evidence supporting any possible correlation implying any kind of causation effect. However if any kind of link is discovered, it does mean disastrous implications on human health. Since the information about the same is very confined, no solid ground has been achieved to draw such conclusions, explained the researchers.
‘Persistent Organic Pollutants (POPs) ‘are chemical substances that persist in the environment, bio-accumulate through the food web, and pose a risk of causing adverse effects to human health and the environment. They were profusely used with the introduction of DDT in the 1940s. As the compounds biodegrade slowly, they continue to find their way into the food chain and ultimately into the blood streams of individuals even though many of these toxins were banned many years ago. Additionally, these compounds can persist in body fat for very long periods of time following exposure.
Obesity and genetic factors are already the well proclaimed factors which trigger the onset of the diabetes among the adult population. No clue has been achieved to find the environmental factors such as pollution to contribute for the same.
During the process of the research, an interesting factor which came into light was that obesity and diabetes was absent in people with low concentrations of POPs in their blood. Meaning thereby, such individuals were more likely to bear risk of diabetes if they were thin with high levels of POPs in their blood. Such a relationship was however, in reverse order if they were overweight but with low levels of POPs.
Scientists are of the view that it requires further exploration. This emphasis has been impressed upon with more stress just because the hypothesis that the POPs exposure could cause diabetes is at present focus. This can be base upon the development of specified cell of tissue cultures. This would lead to the development of an effective method to combat the side effects of POPs on the human health.
This article is the property of http://www.Healthpm.com
Copying and publishing any article from our site is strictly NOT allowed.
